The team homed in on a gene called Snhg9, which produces a long, noncoding RNA whose function was unknown. A series of experiments showed that the RNA molecule produced by Snhg9 binds to a protein called CCAR2, setting off a molecular cascade that prevents dietary fats from being absorbed by the cells that line the intestine.
In germ-free mice and mice genetically altered to produce extra Snhg9 RNA, the process kicked off by this molecule protected them from becoming obese, even on a Western-style diet. Additional tests showed these animals weren't absorbing as much dietary fat as wild-type mice. However, mice genetically altered to lack Snhg9 gained excess weight on the high-sugar, high-fat diet, even when their gut microbes were killed off with antibiotics.led by Drs.
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