Covid has hit the ITV show just days before the final, was also closed for the night so it could be disinfected and stop a further outbreak.“The producers have grand plans to make the final bigger than ever so no one is seen without a mask and there have been loads more people queueing up to get themselves tested on site.
“The team also closed down the night shift tent which sells snacks and drinks to the crew in order to give it a deep clean.
Source: Entertainment Trends (entertainmenttrends.net)
Whose got it
High spirits
What a surprise!!
Yawn. Zzzzzzzz
🤣🤣🤣
It’s either the guy with the dingo dollars brought it in on his bike or Matt Hancock contracted it from the sheep vagina.
Care factor = 0
How do you read this, does this mean the winner has been announced and hidden from Joe public. 🤔🤔
Probably Matt Hancock must of cause it lol 😂
STOP scaring the public people with your b********. China at it again
Ps how has someone got Covid if no one left Campus for a week. And everyone been near everyone, I guess it’s all the crap they eat.. lol
Don’t you mean the Flu, there is no Covid ffs. Flu flu flu couching sneezing it’s flu
🤣🤣🤣🤣
From some of the childish food stunts, it's a wonder some haven't gone down with gastro enteritis or some other food poisoning? Wonder when the first accident or serious injury will happen in this stupid party game ❓🙄
😂it will be Hancocks fault no doubt 🥴
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Frontiers | Dysfunctional purinergic signaling correlates with disease severity in COVID-19 patientsEctonucleotidases modulate inflammatory responses by balancing extracellular ATP and adenosine (ADO) and might be involved in COVID-19 immunopathogenesis. Here, we explored the contribution of extracellular nucleotides metabolism to COVID-19 severity in mild and severe cases of the disease. We verified that the gene expression of ectonucleotidases is reduced in the whole blood of patients with COVID-19 and is negatively correlated to CRP plasma levels, an inflammatory marker of disease severity. In line with these findings, COVID-19 patients present higher ATP levels in plasma and reduced levels of ADO when compared to healthy controls. Cell type-specific analysis revealed higher frequencies of CD39+ T cells in severely ill patients, while CD4+ and CD8+ expressing CD73 are reduced in this same group. The frequency of B cells CD39+CD73+ is also decreased during acute COVID-19. Interestingly, B cells from COVID-19 patients showed a reduced capacity to hydrolyze ATP into ADP and ADO. Furthermore, impaired expression of ADO receptors and a compromised activation of its signaling pathway is observed in COVID-19 patients. The presence of ADO in vitro, however, suppressed inflammatory responses triggered in patients’ cells. In summary, our findings support the idea that alterations in the metabolism of extracellular purines contribute to immune dysregulation during COVID-19, possibly favoring disease severity, and suggest that ADO may be a therapeutic approach for the disease.
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