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Alzheimer's disease, the most common form of dementia, is thought to start because of a build-up of two proteins in the brain: amyloid and tau.
This cascade of events has been known for many years and is how the disease progresses in most people with Alzheimer's.A recent study in the journalinvestigated whether our genes might influence how resilient we are against Alzheimer's disease symptoms when there are high levels of amyloid in our brain.
The last group, they considered as being resilient to Alzheimer's disease since they had the proteins in their brains but did not have the symptoms or a diagnosis of Alzheimer's disease during their lifetime. Is there a way we can make ourselves more resilient to developing Alzheimer's disease regardless of our genes?and drugs allow us to reduce our risk of developing Alzheimer's disease in the future.
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