In the fight against Alzheimer's, researchers have developed a promising, preventative therapeutic approach. They specifically targeted the amyloid beta biomolecule, which triggers the hyperactivity of nerve cells typical of the brain disease in its early stages.
In the fight against Alzheimer's, researchers have developed a promising, preventative therapeutic approach. They specifically targeted the amyloid beta biomolecule, which triggers the hyperactivity of nerve cells typical of the brain disease in its early stages. They succeeded in developing and using a protein drug that can suppress the effects of the harmful molecule.
The results obtained on mice in the laboratory indicate that neuronal dysfunctions could even be repaired. The study was published in the journal. The researchers hope that the protein they investigated, which experts refer to as amyloid-beta-binding anticalin , can halt the progression of the serious neurodegenerative disease at an early stage.
Dr. Benedikt Zott emphasizes:"We are still a long way from a therapy that can be used in humans, but the results in animal experiments are very encouraging. The effect of completely suppressing neuronal hyperactivity in the early stages of the disease is particularly remarkable." It is still unclear whether the effect can actually be achieved in human patients outside the laboratory. In any case, a more effective form of administration of the active ingredient is currently being developed. In 2016, the active substance solanezumab, which was supposed to have a similar effect, proved to be a failure in large-scale clinical trials, but this can be explained by its different molecular structure.
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