Investigating the alveolar type II reparative response in COVID-19 pneumonia JIDJournal UMNews covid COVID19 SARSCoV2 pneumonia covidpneumonia COVIDpneumonia
By Dr. Chinta SidharthanJun 9 2023Reviewed by Lily Ramsey, LLM In a recent study published in The Journal of Infectious Diseases, researchers examined the response of alveolar type II pneumocytes to tumor necrosis factor alpha and Bruton tyrosine kinase during coronavirus disease 2019 pneumonia.
The development of effective treatments to prevent severe COVID-19 is based on understanding the underlying mechanisms in the progression of COVID-19 pneumonia. Related StoriesThe reparative process involving ATII cells also indicates that for the SARS-CoV-2 infection to progress to COVID-19 pneumonia, SARS-CoV-2 can significantly reduce the ATII cell population by increasing viral replication in many susceptible cells.
Results The results reported that during COVID-19 pneumonia, ATII cells, whether uninfected or infected with SARS-CoV-2, succumb to necroptosis induced by TNF-α, pyroptosis induced by BTK, and PANoptosis — a hybrid inflammatory death cell form where distinctive pathologies related to COVID-19 are generated in ATII cells by a PANoptosomal latticework.
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